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简历怎么写:PAR蛋白质

简历怎么写:PAR蛋白质

PAR蛋白质坚决不像他们可以绑定到皮层快速交换和横向扩散与胞质池。借助复杂网络交互,极性是保持平衡的动态行为标准蛋白质(Schulze & Schierenberg,2011)。在维护阶段,Par 2中起着重要作用。防止攻击的复杂的前部向后部,重新装配的杆阻碍后的肌动球蛋白疫源地。它将触发异常后的直接流动和重新分配的PAR 3-PKC 3 PAR 6 t后皮层(波尔&包,2010)。它所在PAR-1后皮层和排除前复杂的从票面1 PAR 2域。极性的维护、PAR 5也参与其中。

简历怎么写:PAR蛋白质
PAR 5结合磷酸化PAR 1从3 PAR或消除前,也不包括后。极性维护机制依赖于小GTPase CDC-42,必将促进6定位在大脑皮层。它还包括前复杂的稳定表示在维护阶段(Noatynska &得,2012)。因此,在维护阶段的结束,胞质分裂沟进入胚胎的一半后使细胞命运的决定因素划分成不同的子细胞。为目的的实现机制,票面蛋白质使控制有丝分裂纺锤波的位置(大梁,2005)。这是机制是成熟后的胚胎。

简历怎么写:PAR蛋白质

The PAR proteins are firmly not bound to the cortex as they can rapidly exchange and diffuse laterally with the cytoplasmic pool. With the help of complex network interactions, the polarity is maintained which balances the dynamic behaviour of the PAR proteins (Schulze & Schierenberg, 2011). In the maintenance phase, Par 2 plays a significant role. To prevent the attack of the complex anterior towards the posterior, the PAR-2 obstructs the re-assembly of the actomyosin foci in posterior. It will trigger the direct flow of abnormal posterior and redistributes the PAR 3-PKC 3-PAR 6 t posterior cortex (Pohl & Bao, 2010). It localizes the PAR-1 posterior cortex and excludes the anterior complex from PAR 1 PAR 2 domain. In the polarity maintenance, PAR 5 is also involved.

简历怎么写:PAR蛋白质
PAR 5 combines the phosphorylated PAR 1 to eliminate it from PAR 3 or anterior and also excluding it from posterior.The polarity maintenance mechanism is dependent on the small GTPase CDC-42 that is bound to the PAR-6 for the promotion of localization which is at the cortex. It also includes the stabilization of the anterior complex which is indicated during the maintenance phase (Noatynska & Gotta, 2012). Therefore, at the ending of the maintenance phase, the cytokinesis furrow enters into the half posterior of the embryo which enables the separation of the cell-fate determinants into the daughter cells. For the purpose of achieving the mechanism, the PAR proteins enable the control over the position of the mitotic spindles (Sommer, 2005). This is the mechanism which is involved after the maturing of the embryo.